1st International Conference on
Neuroscience, Neuroinformatics, Neurotechnology and Neuro-Psycho- Pharmacology

1st International Conference on Neuroscience, Neuroinformatics, Neurotechnology and Neuro-Psycho-Pharmacology

We kindly invite you to participate in the 1st International Conference on Neuroscience, Neuroinformatics, Neurotechnology and Neuro-Psycho-Pharmacology organized by the Romanian Academy - Romanian Group for Brain Research, Romanian Society of Neurology, the Foundation of the Romanian Society of Neurology, the National Neuroscience Society of Romania and the Romanian Society for Automation and Technical Informatics.

The aim of the conference is to bring together experts from various areas of brain science, neuroscience, clinical neurology, psychology, pharmacology with engineers in systems science, neuroinformatics, computer science and technology in order to debate aspects of the brain and mind, to identify efficient theories, models and tools that will contribute to a better understanding of the brain and its functioning. The conference will be organized in Bucharest on 15-18 November 2018. The opening ceremony and the invited papers will be presented in the main Hall of the Romanian Academy.

Call for papers


Romanian Academy - Romanian Group for Brain Research
Romanian Society of Neurology
Foundation of the Romanian Society of Neurology
Romanian Society for Automation and Technical Informatics
National Neuroscience Society of Romania

General Chair: Ioan Dumitrache

General Co-Chair: Ovidiu Bajenaru

General Co-Chair: Luiza Flonta

General Co-Chair: Leon Zagrean


The aim of 1st International Conference on Neuroscience, Neuroinformatics, Neurotechnology and Neuro-Psycho-Pharmacology is to cover areas in specific topics listed below:

1. Neurocognitive domains
2. Neural mechanisms of human behavior
3. Neuroelectrophysiology and neuroimagery in the research of human brain functioning
4. Stroke lesions as models of understanding human brain functions – advantages and limits
5. Quantitative theories and modeling of brain functions
6. Cellular Neurobiology
7. New technologies for brain computer interfaces
8. Models, theories and infrastructures in brain data analysis
9. Machine perception
10. Neurobiotics
11. Neuroinformatics
12. Qualia and Phenomenal Consciousness


* Paper Submission: September 15, 2018
Paper Submission: July 31, 2018

Paper Submission: June 30, 2018
* Author Notification: October 10, 2018
* Author Registration: November 10, 2018
* Conference venue: November 15-18, 2018


Jon H. Kaas

Centennial Professor, Department of Psychology
Vanderbilt University, USA
Member in National Academy of Science of USA
Member in American Academy of Arts and Sciences

Presentation: The organization of parietal-frontal sensorimotor networks in primate brains

Compared to other mammals, primates have a large number of visual areas in neocortex, as well as expanded posterior parietal cortex and premotor and prefrontal areas of cortex. Visual areas that directly or indirectly project to posterior parietal cortex are considered to be in the dorsal stream of cortical networks for selecting and producing complex behaviors. Our research in monkeys and prosimian galagos indicates that a large part of posterior parietal cortex is subdivided into eight or more small regions or domains where electrical stimulation evokes a specific but different action such as reaching, grasping, running, or looking. Each domain of posterior parietal cortex selectively activates functionally matched domains in motor and premotor cortex, where the motor domains are necessary components of the functional networks. We regard the three sets of interconnected domains as three levels of decision making, where sensory information, cognitive and memory components, and motor planning sources of information successfully influence the motor outcome. The nature of the fractured motor maps in premotor and motor cortex suggest that their domains include a mixture of functionally different microcolumns that together reflect the functions of the domains.

Nobuyuki Nukina

Professor, Head of the Laboratory of Structural Neuropathology,
Doshisha University Graduate School of Brain Science

Presentation: Insoluble proteins and neurodegeneration: deposition and transmission

Neuropathological hallmarks of neurodegenerative disorders are depositions of insoluble proteins such as Abeta and tau in Alzheimer disease, alpha-synuclein in Parkinson disease, and polyglutamine in Huntington disease (HD). p62 is important regulatory proteins for selective autophagy, by which aggregated proteins are degraded, and is associated with several inclusions of neurodegenerative disorders including nuclear inclusions of HD. We have been focusing on the protein quality control system and its role on clearance of polyglutamine aggregates. In this talk, I will introduce those works and also the effect of p62 depletion on the polyglutamine mouse models. The results suggest that the genetic ablation of p62 in HD mice models enhanced cytoplasmic inclusion formation due to affected autophagic clearance of polyQ inclusions, decreased their nuclear influx and ameliorates paradoxically the disease phenotypes by decreasing toxic nuclear inclusions. Because the autophagic clearance system works in cytoplasm, this system should work more in other neurodegenerative disorders with cytoplasmic inclusions. To detect the effect of factors on the protein deposition, we established prion-like transmission experiments using alpha-synuclein seeds in vivo. Unexpectedly, the seeds disseminated very rapidly in vivo. I will discuss about the significance of this phenomenon.

Vance P. Lemmon

Professor, The Miami Project to Cure Paralysis, Department of Neurological Surgery,
Center for Computational Science, University of Miami Miller School of Medicine, Miami, USA

Presentation: Using machine learning, phenotypic assays and biochemical profiling to identify drug targets and anti-targets to promote axonal sprouting and regeneration.

Phenotypic assays on live cells are efficient at finding compounds that produce desired cellular outcomes, but it is often impossible to identify the molecular targets of these compounds. Biochemical binding assays, in contrast, are effective at identifying compounds that bind to targets, but such compounds may not work in live cells or may cause undesirable "off target" effects. By using libraries of compounds that have been screened on hundreds of enzymes (for example kinases) in phenotypic assays, it is possible to link these two approaches. Machine learning can then be used to identify targets (kinases that when inhibited enhance the desired phenotypic outcome) and anti-targets (kinases that when inhibited antagonize the desired phenotypic outcome). This drug discovery pipeline also uncovers synergies among targets that can dramatically increase compound efficacy. We have used our pipeline to identify kinase targets that can be inhibited to promote axon initiation, axon extension and axon branching. Compounds that inhibit multiple molecular targets in vitro have proven effective at promoting regeneration in vivo.

Mavi Sanchez-Vives

Research Professor ICREA
IDIBAPS Institut d'Investigacions Biomediques August Pi i Sunyer, University of Barcelona, Spain

Presentation: Impact of Virtual Embodiment on Sensory and Motor Processing

Our stable body representation can be challenged not only by neurological conditions but also by body transformation illusions such as the rubber hand illusion. In the last decade we have demonstrated that providing congruent sensorimotor correlations, we can induce the illusion that a virtual body in immersive virtual reality is our own body. The experience of "embodiment" of a virtual body has consequences that include physiological, behavioural, and psychological ones. Here I will discuss the impact of virtual embodiment on sensory processing including pain perception, concluding that virtual reality can be used to reduce chronic pain. Further, a virtual body can be used to better understand motor processing. I will explain the brain signals associated to motor errors and how controlling a virtual body through a brain computer interface can help us to understand the brain mechanisms of agency over movements. Virtual bodies become then a tool in neuroscience to study brain function and also a promising strategy in neurorehabilitation.

Michael Chopp

Vice-Chairman Department of Neurology
Scientific Director of the Neurosciences Institute
Zoltan J. Kovacs Chair in Neuroscience Research at Henry Ford Health System
Distinguished Professor of Physics at Oakland University

Presentation: Exosomes-Biological Nanoparticles For The Treatment Of Neurological Disease And Injury

Exosomes are nanovesicles (~30-120nm) that mediate intercellular communication in many organisms. They contain proteins, mRNA, miRNA, lipids and DNA, which can be transferred to and thereby alter recipient cells. Cell-based therapies induce neurovascular plasticity and promote neurological recovery from neural injury and degenerative disease via release of exosomes. Therefore, exosomes harvested from parent cells employed as cell-based therapies may be used to treat injury and disease, and these exosomes may provide a safer and more effective means to enhance neuroplasticity and thereby neurological recovery than stem cells. Here, I will provide an introduction into the biology and an overview of some therapeutic applications for exosomes in mediating neuroplasticity and thereby neurological recovery after neural injury and neurodegenerative disease. The vital role of exosomes in intercellular communication, with particular emphasis on their microRNA content as master post transcriptional gene regulators will be described. A primary focus of this presentation will be on the use of exosomes harvested from various cell sources as a means to remodel the nervous system after onset of disease or injury. A major leap forward in the implementation and development of exosome based therapeutics is the therapeutic use of "designer" exosomes. These "designer" exosomes are, enriched with specific microRNAs, with the miRNA content tailored to specifically enhance the therapeutic restorative effects of exosome treatment for a targeted disease, whether stroke, traumatic brain injury or peripheral neuropathy. Use of exosomes for the neurorestorative treatment of cerebral injury in a variety of species, including non-human primates, pigs and rodents, and the utility of exosomes from different parental sources to treat experimental preclinical models of dementia and neurodegenerative diseases will also be described. Since exosomes mediate intercellular communication our ability to regulate and alter intercellular and inter-organ interaction via exosomes provides a powerful potential therapy for many diseases

Ioan Opris

Associate Professor, University of Miami, Milller School of Medicine,
Miami Project to Cure Paralysis, Center for Neuroengineering, and
Department of Biomedical Engineering, Miami, Florida, USA
Professor, USAMV, School of Veterinary Medicine, Bucharest, Romania

Presentation: The Emergence of the Mind

The prefrontal cortex (PFC) of human and animal brain has been postulated to play critical roles in the executive control function, while the Hippocampus is instrumental in the formation of long-term memories and the Brainstem in the control of behavior. To gain insights into the neurobiological mechanism of such cognitive functions, one needs to understand the input-output transformational properties of the PFC, hippocampal and brainstem's neuronal micro-circuits. This implies that spike trains of columnar neuronal ensembles of neurons are recorded with a biomorphic multi-electrode array from the prefrontal cortical layers of animals performing a cognitive task and then stimulated with appropriate patterns of electrical pulses. This modular approach provides clues about how the input spike trains are transformed into output spike trains by the PFC and hippocampal micro-circuitry. Recent results demonstrate the following integrative aspects: 1. Functional integration of perceptual and behavioral signals across cortical layers during executive control. The integrative effect of dlPFC minicolumns was shown by: (i) increased correlated firing on correct vs. error trials; (ii) decreased correlated firing when the number of non-matching images increased; and (iii) similar spatial firing preference across cortical-striatal cells during spatial-trials, and less on object-trials. 2. Causal relations to integration of cognitive signals by the minicolumnar turbo-engines. The inter-laminar integration between the perceptual and executive circuits was facilitated by stimulating the infra-granular layers with firing patterns from supra-granular layers that enhanced spatial preference of percent correct performance on spatial trials. 3. Integration across hierarchical levels of the brain. This functional connectivity may have important implications for the understanding of functional integration within the PFC of the cognitive functions supporting the emergence of mind

Mihai Moldovan

Termobit Prod SRL, Bucharest, Romania,
Division of Physiology and Neuroscience,
"Carol Davila" University of Medicine and Pharmacy, Bucharest, Romania;
Associate Professor, Department of Neuroscience
University of Copenhagen, Denmark

Presentation: Probing awareness using EEG reactivity measures

Recording the electrical activity of the brain by means of electroencephalography (EEG) can reliably distinguish wakefulness from sleep. Nevertheless, the distinction of awareness (content of consciousness) from arousal remains challenging using only spontaneous EEG recordings. Quantification of EEG changes to standardized stimulation paradigms is referred to as EEG reactivity. My lecture will review the EEG reactivity work carried out in the ComaEEG.RO consortium comprised of an increasing number of basic scientists, clinicians and industry partners. Our central hypothesis is that the brain activity, as reflected by its EEG signature, can be described as a sequence of task-oriented stimulus processing frames alternating with default mode frames devoid of task-demand. Such an alternating pattern can be directly observed in deep comatose states where the EEG shows large bursts of stimulus-evoked activity alternating with periods of flat EEG, consistent with the suppression of default frames commonly associated with consciousness. We developed a method to distinguish default activity frames in both burst-suppression and continuous EEG referred to as default EEG reactivity (DER, patent pending). In brief, we segmented the multi-channel EEG into consecutive classes with similar topographic frequency distribution and then identified the class with the largest decrease in occurrence probability during 1-minute stimulation epochs. To distinguish awareness, we focused on a particularly salient stimulus, the subjects own name, whereas the level of arousal was controlled by measures of heart rate variability. Our data suggest that DER can be used to derive measures of awareness with both clinical and experimental applications.

Monica Baciu

Laboratoire de Psychologie et Neurocognition,
UMR 5105 CNRS ,
Universite Grenoble Alpes

Presentation: Neuroplasticity Of Cognitive Networks In Patients With Temporal Lobe Epilepsy.A Multimodal And Integrative Approach.

The interaction between language and memory is obvious in the daily life, for instance, when trying to lead a conversation properly, as we need memory to keep track of the flow of conversation. This interaction is also notable when a brain lesion arises in a specific area devoted to language for instance, and symptoms appear in the memory domain, frequently observed in neurological, neuropsychological and psychiatric disorders. Language and memory mechanisms and cerebral networks are well studied separately, but less known in interaction. A better knowledge of this interaction is fundamental from a neurocognitive standpoint, given that cognitive processes cannot be considered anymore as monolithic functions with modular representation, but rather in interaction, based on common brain substrates and "hub" regions. The interaction is also important from a clinical perspective (i) to understand the potential contribution of each function to a specific cognitive deficit, (ii) to be more accurate when it comes to anticipate the cognitive deficit after brain-lesions or after surgery and (iii) to develop neuro-rehabilitation tools. Overall, our work sets the goal of providing new insights into the study of interactive language-andmemory models considered dynamically through the neuroplasticity, by taking advantage of a specific neurological condition, the temporal lobe epilepsy presenting pharmaco-resistance and requiring surgery. To reach our goals, we use a multimodal methodology to collect a variety of data and biomarkers. Together with cognitive scores measured in patients, the multimodal data will be integrated into analyses that allow to assess multimodal networks for language-and-memory. An auxiliary clinical goal of our work is to provide statistically-determined robust data of preoperative and postoperative patterns of functional language-and-memory reorganization, objectify relationships between pre-surgical and post-surgical datasets and develop tools able to anticipate the cognitive outcome after surgery.

Karl J. Friston

Professor, MB, BS, MA, MRCPsych, FMedSci, FRSB, FRS
Wellcome Principal Fellow
Scientific Director: Wellcome Trust Centre for Neuroimaging
Institute of Neurology, UCL

Presentation: I am therefore I think

This overview of the free energy principle offers an account of embodied exchange with the world that associates neuronal operations with actively inferring the causes of our sensations. Its agenda is to link formal (mathematical) descriptions of dynamical systems to a description of perception in terms of beliefs and goals. The argument has two parts: the first calls on the lawful dynamics of any (weakly mixing) ergodic system - from a single cell organism to a human brain. These lawful dynamics suggest that (internal) states can be interpreted as modelling or predicting the (external) causes of sensory fluctuations. In other words, if a system exists, its internal states must encode probabilistic beliefs about external states. Heuristically, this means that if I exist (am) then I must have beliefs (think). The second part of the argument is that the only tenable beliefs I can entertain about myself are that I exist. This may seem rather obvious; however, it transpires that this is equivalent to believing that the world - and the way it is sampled - will resolve uncertainty about the causes of sensations. We will consider the implications for functional anatomy, in terms of predictive coding and hierarchical architectures in the brain. We will conclude by looking at the epistemic behaviour that emerges using simulations of active inference, with a special focus on visual foraging.


Hidehiro Mizusawa

MD, PhD, President, National Center of Neurology and Psychiatry,
Professor Emeritus, Tokyo Medical and Dental University

Presentation: Prism adaptation test (PAT) - A new quantitative test of cerebellar motor learning

Spinocerebellar degeneration usually progress very slowly and changes of scores of clinical scales like SARA are quite small. Therefore, we need a practical test which can quantitatively evaluate subtle changes of cerebellar functions. Among various functions of the cerebellum, motor learning has a long history of extensive researches. We thought prism adaptation using finger reach movement may be a good task because adaptation time is fairly short during the task. An examinee wearing prism-equipped goggles touches the index finger to the target on a touchscreen in every trial. The whole test was composed of 3 consecutive sessions: 1) 50 trials with normal vision (BASELINE), 2) 100 trials with a prism shifting the visual field 25 ˚ rightward (PRISM) and 3) 50 trials without the prism (REMOVAL). In normal control, touched points deviated greatly with a prism but gradually returned to the correct target during repeated trials. The touched points deviated similarly to the opposite direction when the prism was removed and returned again to the correct target after repeated trials. Adaptation index (AI) was calculated by multiplying each probability of acquisition in the last 10 trials of PRISM, retention in the initial 5 trials of REMOVAL and extinction of in the last 10 trials of REMOVAL. AI clearly distinguished patients with cerebellar ataxia from normal controls and was significantly correlated with SARA and 9HPT scores. This system is compact and easy to use at outpatient clinics and useful to quantitatively evaluate the cerebellar function in clinical trials for cerebellar disorders.

Javier DeFelipe

Research Professor, Instituto Cajal (CSIC)
Laboratorio de Circuitos Corticales (CTB)
Universidad Politecnica de Madrid,
Madrid, Spain

Presentation: Brain connectomics: From Cajal to present

From the outset of Cajal's studies in 1888 with the method of Golgi he provided support for his belief that dendrites and axons end freely in the nervous system and that they communicate by contact. This hypothesis contrasted with the most prevalent idea at the time that the elements of the nervous system formed a continuum. The existence of a continuous network would more readily explain the flow of currents, but how could this be possible through an infinitely interrupted and fragmented nervous system? Cajal proposed that neurons showed a morphological and functional polarization in such a way that neurons could be divided in general into three distinct regions: a receptor apparatus (dendrites and soma), the emission apparatus (the axon) and the distribution apparatus (terminal axonal arborisation). Cajal's new ideas about the connections between neurons led to novel theories on the relationship between neuronal circuits and brain function. Indeed, this hypothesis gave rise to a new era in neuroscience and to the tracing of the first point-to-point connectivity maps. In the 1930s, it had been shown histologically that the terminal axonal bouton was separated by a "membranous synaptic barrier". At the areas of contact between the axon terminal and the soma or dendrite, only one membrane was visible (the synaptic membrane), presumably since the membrane of the pre- and post-synaptic elements were so close together that only a single membrane could be distinguished. Thanks to the introduction of transmission electron microscopy (TEM) in the 1950s the nature of synapses was examined, confirming a critical aspect of the neuron theory: the pre-synaptic and the post-synaptic elements are physically separated by a space about 20 nm wide, the synaptic cleft. The recent development of automated EM techniques (e.g., FIB/SEM microscopy) will represent a true revolution in the examination of synaptic circuits.


Mikhail Lebedev

Research Scientist, Department of Neurobiology
Duke University Medical Center,
Durham, USA

Presentation: Repairing the brain with brain-machine interfaces

The lecture will cover the major advances made in brain-machine interfaces (BMIs). BMIs link the brain to external devices, with an eventual goal of recovery of motor, sensory and cognitive functions to patients with neurological conditions. Over the past half-century, BMIs have advanced significantly from the early ideas that sounded like science fiction to the modern high-tech implementations. In particular, intracranial recordings using multichannel implants have enabled real-time control of artificial limbs by nonhuman primates and human subjects. BMIs can restore upper-limb and lower-limb functions. Furthermore, bidirectional BMIs can provide artificial sensory feedback, allowing users to perceive the movements of prosthetic limbs and their interaction with external objects. BMIs can also multitask, like simultaneously decoding orientation of spatial attention and motor goals. Recently, BMI approach was employed to build brain-nets that enable information exchange between individual brains and execution of cooperative tasks. Overall, BMIs appear to be an efficient approach to augmenting the brain function, with limitless perspectives.


Valentin Dragoi

Levit Distinguished Professor of Neuroscience
Dept. of Neurobiology and Anatomy
McGovern Medical School
Univ. of Texas-Houston

Presentation: Population synchrony in cortical networks

Brain activity during wakefulness is characterized by fluctuations in neuronal responses at different time scales. Whether these fluctuations play any role in modulating the coding of sensory information and the accuracy of behavioral responses is poorly understood. Using multiple-electrode recording techniques I will show that slow changes in local population synchrony in monkey visual cortex impair the coding of sensory information and perceptual performance. These changes also occur in executive areas, such as prefrontal cortex, while monkeys freely explore their environment during foraging. However, while population synchrony is detrimental for neural coding and behavior at long time scales, it plays a beneficial role at shorter time scales. Indeed, by simultaneously recording visual cortical populations in multiple areas we recently discovered that the precise temporal coordination between the spikes of three of more neurons carries information about perceptual reports in the absence of firing rate modulation. These results demonstrate an unexpected functional impact of synchrony in local cortical networks at slow and rapid time scales.

Angelo Antonini

Department of Neuroscience
University of Padua
Padua Italy

Presentation: Neuroimaging Correlates Of Cognitive And Behavioral Dysfunction In Parkinson's Disease

Parkinson's disease (PD) is a multi-dimensional disorder characterized by motor and non-motor features including cognitive dysfunction. Dementia prevalence is about the 80% in patients with 15-20 years of disease duration affecting quality of life for both patients and careers. However only the 25% of PD with dementia (PDD) is recognized during clinical practice. There is an urgent need to identify preclinical PDD markers to implement therapeutic and supportive strategies at a stage of the disease when they are more likely to have efficacy. Evidence suggests that mild cognitive impairment (MCI) in PD represents a risk factor for dementia. The attempt to standardize cognitive criteria for MCI did not help definition of a preclinical cognitive pattern, mainly due to lack of an adequate neuropsychological battery and valid cut-off norms making the frequency of MCI diagnosis heterogeneous. An adequate clinical approach would be to first frame the cognitive problems by an unstructured interview and explore if there is insight of the interference in daily life. A more detailed neuropsychological evaluation is needed to explore performance in specific cognitive domains. It is vital that cognitive assessment is undertaken by a qualified clinical neuropsychologist. Neuroimaging can help defining the anatomical and functional correlates of cognitive defects. This can be achieved either by structural MRI or functional imaging.

Hari Shanker Sharma

Director, Int. Exp. CNS Injury & Repair (IECNSIR);
Professor of Neurobiology (MRC); Docent in Neuroanatomy (UU)
Uppsala University, University Hospital, Anesthesiology & Intensive Care Medicine, Dept. Surgical Sciences
President, International Association of Neurorestoratology (IANR);

Presentation: Novel therapeutic strategies using nanomedicine

Abstracts and biography are available here



Ioan Dumitrache, University Politehnica of Bucharest, Romania
Ovidiu Bajenaru, University of Medicine and Pharmacy Carol Davila, Bucharest, Romania
Luiza Flonta, University of Bucharest, Romania
Leon Zagrean, University of Medicine and Pharmacy Carol Davila, Bucharest, Romania

International Program Committee

Alexandru Babes, University of Bucharest, Romania
Rodica Balasa, University of Medicine and Pharmacy of Targu Mures, Romania
Theodore W. Berger, USC Viterbi School of Engineering
Simona Iuliana Caramihai, University Politehnica of Bucharest, Romania
Javier DeFelipe, Universidad Politecnica de Madrid
Valentin Dragoi, University of Texas Health Science Center, SUA,
Mircea Dumitru, University of Bucharest, Romania
Jon H. Kaas, Vanderbilt University School of Medicine
Mikhail Lebedev, Duke University, USA
Hidehiro Mizusawa, National Center of Neurology and Psychiatry, Japan
Mihai Moldovan, University of Copenhagen-Department of Neuroscience and Pharmacology, Denmark
Dafin Muresan, University of Medicine and Pharmacy Iuliu Hatieganu, Cluj-Napoca, Romania
Nobuyuki Nukina,Ph.D.,M.D. Laboratory of Structural Neuropathology, Doshisha University
Ioan Opris, University of Miami, Miller School of Medicine, Florida, USA
Nirvana Popescu, University Politehnica of Bucharest, Romania
Stefan Trausanu-Matu, University Politehnica of Bucharest, Romania
Catalina Tudose, Carol Davila University of Medicine and Pharmacy, Bucharest, Romania
Victor Voicu, University of Medicine and Pharmacy Carol Davila, Bucharest, Romania

National Organising Committee

Mihnea Moisescu, University Politehnica of Bucharest, Romania
Ioan Sacala, University Politehnica of Bucharest, Romania
Octavian Arsene, University Politehnica of Bucharest, Romania
Marius Leordean, University Politehnica of Bucharest, Romania
Razvan Radu, University of Medicine and Pharmacy Carol Davila, Bucharest, Romania
Beatrice Radu, Faculty of Biology University of Bucharest, Romania


To be announced


Authors are invited to submit abstracts written in English.
All submissions will be reviewed based on originality, significance, quality and clarity.
The Program Committee will select the contributions after rigorous peer-review.
Selected contributions will be invited for oral or poster presentation.
The Program Committee will select relevant contribution and authors will be invited to submit a full paper to be published in a special issue of the Romanian Journal of Neurology - http://rjn.com.ro/
Please send your abstract containing the title of the paper, authors and affiliation, corresponding author, key words and no more than 500 words, A-4 sheets, Times New Roman font. Only PDF format is accepted for submissions. Details about author(s) must be provided: first name, last name, affiliation, mailing address, e-mail address. For papers with multiple authors the contact person must be indicated.
The conference uses EasyChair platform for the paper submission. In the submission phase, please select the main topics related to your paper, specify the full name for all authors and include the keywords. The conference submission system is available at:


All accepted papers MUST be registered by one of the authors.
The registration deadline and the payment proof deadline is November 10, 2018.
Registration fee for the conference is 250 Euros and includes participation, Conference proceedings and CD, coffee breaks and cocktail.
Before 10th of November early registration is 200 Euros. The fee for PhD students is 100 Euro (a scanned proof will be needed)
Registration for students, master students and medical residents is free. (a scanned proof will be needed)
Begistration fee can be payed in EUR or in RON (using the BNR excahnge rate valid for the day of the payment) by bank tranfer using the following bank account information. Please specify "ICNNN coference tax" followed by paper ID and main / corresponding author name in details section.
Please send a proof of paiment to the following email address veni@rotravelplus.com.
For Payments in RON:
Titular cont: Romania Travel Plus
Banca ING
Cont: RO05 INGB 0001 0001 3596 8938
For Payments in Euro:
Account holder: Romania Travel Plus
ING Bank
RO94 INGB 0001 0001 3596 0740


Conference venue:
World Trade Center
New Yourk Hall - link

Romanian Academy
Aula of the Romanian Academy - link

For travel arrangements and hotel booking please contact:
Romania Travel Plus - contact person: Venita Doana
e-mail: venita.doana@rotravelplus.com / Phone: 0372732624

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